Fig. 6
From: Neurobiological effects of gallic acid: current perspectives
Overall neurological activities of gallic acid against different diseases and states of different mediators or proteins. ↑increase, ↓decrease. Aβ amyloid β protein, TNF- α tumour necrosis factor-α, TBARS 2-thiobarbituric acid reactive substances, κ-CN kappa-casein, NF-kB nuclear factor kappa B, GSH glutathione, MDA malondialdehyde, TCA tricarboxylic acid, 5-HT1A 5-hydroxytryptamine (serotonin) receptor 1A, DP dopamine, AChE acetylcholinesterase, GABA gamma-aminobutyric acid, COX-2: cyclooxygenase-2, IL-6 interleukin-6, IL-10 interleukin-10, IL-1β interleukin-1β, ROS reactive oxygen species, MPO myeloperoxidase, RIPK-1 receptor-interacting protein kinase-1, RIPK-3 receptor-interacting protein kinase-3,NO nitric oxide, iNOS inducible nitric oxide synthase, LPO lipid peroxidation, MCP1 monocyte chemoattractant protein-1, HPO hydrogen peroxide, AND adrenaline; PN plasma nitrile, NOS nitric oxide synthase, CoS corticosterone